Contribution of Intimal Smooth Muscle Cells to Cholesterol Accumulation and Macrophage-Like Cells in Human Atherosclerosis

By S. Allahverdian, Ali Cyrus Chehroudi, B. McManus, T. Abraham, G. Francis. Circulation, 2014

639 citations recorded by the source corpus.

Subfields: Immunology, Surgery

View Paper: https://www.semanticscholar.org/paper/5e9995c3049c7f0bdc3ec6cb02d026c3c84e9d49

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In Immunology

1. G. Owens
2. I. Tabas
3. A. Lichtman

Citations in the corpus

• Pathophysiology of Atherosclerosis (2022, background): “Indeed, their contribution to the sum of foam cell population within the plaque is significant [20].”
• Vascular smooth muscle cells in atherosclerosis (2016, background): “However, recent studies showed that 40% of foam cells within advanced human coronary artery lesions express both the SMC marker ACTA2 and the macrophage marker CD68, although it is unclear if these represent VSMC-derived cells that have activated macrophage markers, are macrophages that have activat”
• Role of smooth muscle cells in vascular calcification: implications in atherosclerosis and arterial stiffness (2018, background): “VSMCs differentiate not only into osteoblasts and chondrocytes during AIC but also into lipid-loaded, foam cell-like macrophages contributing directly to the advanced atherosclerotic plaque progression.(29,97,98) In a confocal microscopy study of human coronary artery sections from”