Mitochondrial dysfunction in NASH: causes, consequences and possible means to prevent it.

By Karima Begriche, Anissa Igoudjil, D. Pessayre, B. Fromenty. Mitochondrion (Amsterdam. Print), 2006

747 citations recorded by the source corpus.

Subfields: Epidemiology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/5c87f0d6d976d82c1bb6467689f471f77cea2e37

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. L. Galluzzi
2. Guido Kroemer
3. Oliver Kepp
4. C. Brenner
5. Beth Levine
6. Douglas R. Green
7. V. Deretic
8. Alec C. Kimmelman

In Molecular Biology

1. Guido Kroemer
2. L. Galluzzi
3. Oliver Kepp
4. C. Brenner
5. Douglas R. Green
6. Beth Levine
7. Thomas G. Cotter
8. Simone Fulda

Citations in the corpus

• Decoding cell death signals in liver inflammation. (2013, background): “Both these functions of autophagy are particularly important in the liver, as various hepatic disorders (i) are accompanied by mitochondrial dysfunction and ROS overproduction [112], and/or (ii) are associated with the accumulation of lipids in the hepatic parenchyma [109,115].”
• Mitochondrial dysfunction: mechanisms and advances in therapy (2024, background): “24,403,404 However, fat deposits gradually vanished after mitochondrial transplantation, along with decreased serum transaminase activity.”