The role of advanced glycation end products in progression and complications of diabetes
By S. Goh, M. Cooper. 2008
1,022 citations recorded by the source corpus.
Subfields: Cell Biology, Clinical Biochemistry, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/5bc73bf41ed24c4a59f50afe51a3183056e2d998
How researchers see this paper
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- Mariyam Khalid
- R. Seiça
- C. Sena
- A. Adem
- A. Pereira
- G. Petroianu
Citations in the corpus
- Endothelial dysfunction - a major mediator of diabetic vascular disease. (2013, background): “11) [191,192], specifically directed to reduce inflammation [193,232] and restore the ox/redox balance in the endothelium may represent promising strategies to ameliorate vascular function in diabetic state.”
- Advanced Glycation End Products and Diabetes Mellitus: Mechanisms and Perspectives (2022, background): “Due to this covalent cross-link formation, the biologically active proteins and enzymes are deactivated [8,16], become resistant to proteolytic digestion [17,18], provide catalytic sites for ROS formation [19], lead to pro-inflammatory events [6], alter intracellular signaling and contribute to seve”