Evaluation of the contributions of ADAMs 9, 12, 15, 17, and 19 to heart development and ectodomain shedding of neuregulins beta1 and beta2.

By K. Horiuchi, Hong-Ming Zhou, Kristine Kelly, K. Manova, C. Blobel. 2005

177 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology, Oncology

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

1. M. Handsley
2. D. Edwards
3. K. Nave
4. C. Pennington

In Oncology

1. M. Handsley
2. D. Edwards
3. B. Trapp
4. K. Nave
5. C. Pennington

Citations in the corpus

• The ADAM metalloproteinases (2008, background): “This therefore represents a fundamental biological signalling mechanism that is conserved from worms to mammals (Horiuchi et al., 2005).”
• Axon-glial signaling and the glial support of axon function. (2008, background): “If the level of axonal NRG1 type III stays below the threshold, the associated axons are not sorted and myelinated but remain grouped together as a Remak bundle with a single nonmyelinating Schwann cell.”