Neuronal KCNQ potassium channels:physislogy and role in disease

By T. Jentsch. Nature Reviews Neuroscience, 2000

845 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Cellular and Molecular Neuroscience, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/585195b9c43c7bfc77e7f611ad5702be2037ad42

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. D. Brown
2. G. Passmore
3. P. Delmas
4. Maarten H. P. Kole
5. G. Stuart

In Cellular and Molecular Neuroscience

1. D. Brown
2. G. Passmore
3. P. Delmas
4. Maarten H. P. Kole
5. G. Stuart

In Molecular Biology

1. D. Brown
2. G. Passmore
3. P. Delmas
4. Maarten H. P. Kole
5. G. Stuart

Citations in the corpus

• Potassium leak channels and the KCNK family of two-p-domain subunits (2001, background): “as KCNQ channels are also suppressed by muscarine and external acidification, and contribute to resting K + flux owing to their activation at hyperpolarized potential”
• Pathways modulating neural KCNQ/M (Kv7) potassium channels (2005, background): “Molecular composition of M channels The molecular composition of M channels defied identification for almost 20 years, but it is now known that they are composed of subunits of the Kv7 (KCNQ) family of K + channel”
• Neural KCNQ (Kv7) channels (2009, background): “5) are expressed in the nervous system (see Jentsch, 2000; Robbins, 2001; Table 1).”