NMDA receptors, place cells and hippocampal spatial memory
By K. Nakazawa, T. McHugh, Matthew A. Wilson, S. Tonegawa. Nature Reviews Neuroscience, 2004
613 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Cognitive Neuroscience, Neurology
View Paper: https://www.semanticscholar.org/paper/579b43dc10517cdb368beb79f37535017a31f849
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cellular and Molecular Neuroscience
- E. T. Rolls
- S. Tonegawa
- T. Kitamura
- Christopher J. MacDonald
- Marc Päpper
- Richard Kempter
- Christian F. Leibold
- Andrea Soltoggio
In Cognitive Neuroscience
- E. T. Rolls
- S. Tonegawa
- T. Kitamura
- Christopher J. MacDonald
- Marc Päpper
- Richard Kempter
- Christian F. Leibold
- Andrea Soltoggio
In Neurology
Citations in the corpus
- Entorhinal–hippocampal neuronal circuits bridge temporally discontiguous events (2015, background): “Studies on genetically engineered mice (Nakazawa et al. 2004) and lesioned rats (Kesner et al.”
- Pattern separation, completion, and categorisation in the hippocampus and neocortex. (2016, background): “…E.T. Rolls / Neurobiology of Learning and Memory xxx (2015) xxx–xxx (as shown by the evidence from CA3-specific vs CA1-specific NMDA receptor knockouts) (Nakazawa et al., 2002, 2003, 2004; Rondi-Reig, Libbey, Eichenbaum, & Tonegawa, 2001).”
- The reorganization and reactivation of hippocampal maps predict spatial memory performance (2010, neutral): “Therefore, it has yet to be demonstrated whether hippocampal representations of goal locations are acquired as a direct result of learning 17 .”
- The mechanisms for pattern completion and pattern separation in the hippocampus (2013, methodology): “…the hippocampus show associative modification as shown by long-term potentiation, and this synaptic modification appears to be involved in learning (see Morris, 1989, 2003; Morris et al., 2003; Nakazawa et al., 2003, 2004; Lynch, 2004; Andersen et al., 2007; Wang and Morris, 2010; Jackson, 2013).”
- Intraneuronal Aβ Causes the Onset of Early Alzheimer’s Disease-Related Cognitive Deficits in Transgenic Mice (2005, neutral): “Because the amyloid and 2 neurofibrillary pathology is predominantly confined to t the hippocampus, cortex and amygdala, learning and a memory were evaluated using the spatial reference vers sion of the Morris water maze (MWM) and inhibitory ( avoidance (IA), which are tasks that are highly dependen”
- Synaptic tagging, evaluation of memories, and the distal reward problem. (2010, background): “for the hippocampus (Nakazawa et al. 2004) or the motor system (Xu et al.”