Auto-antibodies to the receptor tyrosine kinase MuSK in patients with myasthenia gravis without acetylcholine receptor antibodies

By W. Hoch, J. Mcconville, S. Helms, J. Newsom-Davis*, A. Melms, A. Vincent. Nature Medicine, 2001

1,182 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Autoimmune myasthenia gravis: emerging clinical and biological heterogeneity. (2009, background): “The NMJ has three basic components (figure 1): (1) the presynaptic motor nerve terminal, where acetylcholine is synthesised, stored, and released; (2) the synaptic space; and (3) the postsynaptic muscle membrane, which contains the AChRs and the enzyme acetylcholinesterase.”
Clinical correlates with anti-MuSK antibodies in generalized seronegative myasthenia gravis. (2003, background): “The report by Hoch et al. (2001) that a proportion of these patients have serum IgG antibodies Brain 126 ã Guarantors of Brain 2003; all rights reserved Downloaded from https://academic.oup.com/brain/article-abstract/126/10/2304/314486 by guest on 30 July 2018 against the muscle-speci®c kinase…”