Prime role for an insulin epitope in the development of type 1 diabetes in NOD mice
By M. Nakayama, N. Abiru, H. Moriyama, N. Babaya, E. Liu, D. Miao, Liping Yu, D. Wegmann, J. Hutton, J. Elliott and 1 more. Nature, 2005
799 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Genetics, Surgery
View Paper: https://www.semanticscholar.org/paper/53e78b54a4974c3c1994a9d6165d8fcb4d41f544
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Citations in the corpus
- Central role of defective interleukin-2 production in the triggering of islet autoimmune destruction. (2008, background): “Among these genes, those that encode MHC class II, insulin, CTLA-4, and IL-2 have been identified as contributors to the breakdown of central and/or peripheral tolerance, although the precise mechanisms associated with pathogenesis remain to be defined (Chentoufi and Polychronakos, 2002; Nakayama e”