Calcium channel regulation and presynaptic plasticity.

By William A. Catterall, A. Few. Neuron, 2008

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From synapse to nucleus: calcium-dependent gene transcription in the control of synapse development and function. (2008, background): “This local increase in calcium concentration can, depending on the cellular context, result in a number of short-term and long-term synapse-specific alterations, including the insertion or removal of glutamate receptor subunits from the membrane, the alteration of synaptic protein function via postt”
Calcium-dependent PKC isoforms have specialized roles in short-term synaptic plasticity (2014, positive): “One possible mechanism is that PKCg could regulate calcium entry to increase p, because increases in calcium entry are known to contribute to some forms of short-term synaptic plasticity (Catterall and Few, 2008).”
Neuronal calcium signaling: function and dysfunction (2014, background): “One important direct function of CaM is possibly the control of vOCs through binding to channel subunits [94].”