Pathology and genetics of tumours of the lung , pleura, thymus and heart

By W. Travis, E. Brambilla, H. Müller-hermelink. 2004

2,045 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/4f1008ece7b5d56fa90b40e01e0eda8b8467a9aa

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Adi F. Gazdar
A. Shaw
N. Lindeman
W. Travis
H. Asamura
Gregory Riely
T. Mitsudomi
M. Beasley

Citations in the corpus

Unique Clinicopathologic Features Characterize ALK-rearranged Lung Adenocarcinoma in the Western Population (2009, background): “11 Bronchioloalveolar 22 1 (6) 21 (7) Acinar 124 4 (25) 120 (39) Papillary 46 0 (0) 46 (15) Solid 134 11 (69) 123 (40) Solid pattern and >10% signet-ring cells, n (%)* 9 (56) 9 (3) <0.”
The IASLC Lung Cancer Staging Project: Proposals for Coding T Categories for Subsolid Nodules and Assessment of Tumor Size in Part‐Solid Tumors in the Forthcoming Eighth Edition of the TNM Classification of Lung Cancer (2016, background): “(IASLC)/American Thoracic Society (ATS)/European Respiratory Society (ERS) lung adenocarcinoma classification, the World Health Organization (WHO) classification of lung cancer recognized only an in situ (Tis) category for squamous cell carcinoma.(1) As squamous cell carcinoma in situ (SCIS) is not ”
International Association for the Study of Lung Cancer/American Thoracic Society/European Respiratory Society International Multidisciplinary Classification of Lung Adenocarcinoma (2011, methodology): “8,215–218 (4) Molecular markers are an important evolving area in evaluation and management of patients with lung adenocarcinoma.”
Carcinoid Tumors (2008, methodology): “The 2004 WHO classification of lung tumors applies the term DIPNECH to a proliferation of scattered single cells, small nodules (neuroendocrine bodies), or linear proliferations of pulmonary neuroendocrine cells confined to the bronchial and bronchiolar epithelium [32].”