Overlapping neuronal circuits in addiction and obesity: evidence of systems pathology

By Nora D. Volkow, Gene-Jack Wang, J. S. Fowler, Frank Telang. Philosophical Transactions of the Royal Society B: Biological Sciences, 2008

818 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Cognitive Neuroscience, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/4951c9bf557083d273bec8d067bc39bdd9079934

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Citations in the corpus

The tempted brain eats: pleasure and desire circuits in obesity and eating disorders. (2010, neutral): “Human dopamine release in dorsal striatum accompanies craving elicited by viewing food or drug cues (in some studies, more strongly correlated than in ventral striatum) (Volkow et al., 2002; Volkow et al., 2008; Wang et al., 2004a).”
Pro v Con Reviews: Is Food Addictive? (2012, background): “For example, in healthy, normal-weight human subjects, ingestion of palatable food releases DA in the striatum in proportion to the ratings of meal pleasantness (44), while food stimuli activate brain regions that are part of the reward circuitry of the brain (45).”
Dysfunction of the prefrontal cortex in addiction: neuroimaging findings and clinical implications (2011, background): “This is not unexpected, given that these disorders and addiction involve similar compromises in neuronal circuit”
fMRI Reactivity to High-Calorie Food Pictures Predicts Short- and Long-Term Outcome in a Weight-Loss Program (2012, background): “to the proposed circuitry for drugs of abuse (Del Parigi et al., 2003; Volkow and Wise, 2005; Volkow et al., 2008).”
Reward, dopamine and the control of food intake: implications for obesity (2010, neutral): “Based on findings from imaging studies, a model of obesity was recently proposed in which overeating reflects an imbalance between circuits that motivate behavior (because of their involvement in reward and conditioning) and circuits that control and inhibit pre-potent responses [22].”