Infection patterns in barley and wheat spikes inoculated with wild-type and trichodiene synthase gene disrupted Fusarium graminearum.
By C. Jansen, D. von Wettstein, W. Schäfer, Karl-Heinz Kogel, Angelika Felk, F. J. Maier. Proceedings of the National Academy of Sciences of the United States of America, 2005
624 citations recorded by the source corpus.
Subfields: Cell Biology, Molecular Biology, Plant Science
View Paper: https://www.semanticscholar.org/paper/491efa829071b9cd7c82f72538e7c854742e341e
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Citations in the corpus
- Comprehending crystalline β-carotene accumulation by comparing engineered cell models and the natural carotenoid-rich system of citrus (2011, neutral): “graminearum strains carrying loss-of-function mutations in the TRI5 gene, the first step in the trichothecene biosynthetic pathway encoding trichodiene synthase, result in the lack of trichothecene biosynthesis and reduced virulence on wheat and barley (Proctor et al., 1995; Jansen et al., 2005; Bod”
- Impacts of changing climate and agronomic factors on fusarium ear blight of wheat in the UK (2012, background): “DON is thought to play a role in virulence (Proctor et al. 1995; Jansen et al. 2005; Maier et al. 2006) and possibly (along with other trichothecenes) in competition against other fungi.”
- Fusarium pathogenomics. (2013, neutral): “) and maize (Zea mays) (7, 39, 62) but not barley (Hordeum vulgare) (41).”