Pathogen-host interactions in Pseudomonas aeruginosa pneumonia.

By R. Sadikot, T. Blackwell, J. Christman, A. Prince. American Journal of Respiratory and Critical Care Medicine, 2005

838 citations recorded by the source corpus.

Subfields: Molecular Biology, Molecular Medicine, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/489b67dacf83f21f308459a4c8364cb73cabe1db

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

1. S. Gellatly
2. K. Drescher
3. Robert E. W. Hancock
4. Kateryna Selezska
5. B. Bassler
6. Brian K. Hammer

In Molecular Medicine

1. Robert E. W. Hancock
2. Marin Kollef
3. S. Gellatly
4. B. Bassler

In Pulmonary and Respiratory Medicine

1. Robert E. W. Hancock
2. Marin Kollef
3. Kateryna Selezska
4. S. Gellatly

Citations in the corpus

• Pseudomonas aeruginosa: new insights into pathogenesis and host defenses. (2013, background): “The thickened ASL severely impairs the immune response, and the persistent immunological stimulation by the bacteria and/or the inability of the host to control inflammation results in chronic lung inflammation (Sadikot et al., 2005; Williams et al., 2010).”
• The Epidemiology, Pathogenesis and Treatment of Pseudomonas aeruginosa Infections (2012, background): “aeruginosa is intrinsically resistant to many the type III secretion system may be activated.[56] antibacterials, including many β-lactams, the This contact-dependent system enables P.”
• A quorum-sensing inhibitor blocks Pseudomonas aeruginosa virulence and biofilm formation (2013, background): “Proc Natl Acad Sci USA 96(5):2408–2413.”
• Pseudomonas aeruginosa population structure revisited under environmental focus (2010, background): “antibiotic treatments fail because an increasingly large number of strains becomes multiresistant towards antibiotics (Palmer et al., 2005; Sadikot et al., 2005; Sriramulu et al., 2005).”