Opposite Effects of Δ-9-Tetrahydrocannabinol and Cannabidiol on Human Brain Function and Psychopathology
By S. Bhattacharyya, P. Morrison, Paolo Fusar-Poli, R. Martín-Santos, S. Borgwardt, T. Winton-Brown, C. Nosarti, C. Carroll, M. Seal, P. Allen and 10 more. Neuropsychopharmacology, 2010
688 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Cognitive Neuroscience, Pharmacology
View Paper: https://www.semanticscholar.org/paper/44e8362ff01bfdb320b7e76c1eb22881d87425f4
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cellular and Molecular Neuroscience
In Cognitive Neuroscience
In Pharmacology
Citations in the corpus
- Cannabidiol enhances anandamide signaling and alleviates psychotic symptoms of schizophrenia (2012, background): “mental studies show that cannabidiol reduces psychosis-like effects ofD(9)-tetrahydrocannabinol and synthetic analogs.(22,23)”
- Cannabidiol: Pharmacology and potential therapeutic role in epilepsy and other neuropsychiatric disorders (2014, background): “In healthy subjects, the acute psychotomimetic effects of D(9)-THC correlated significantly with attenuation of striatal activation during a verbal memory task, whereas CBD augments striatal activation in the same task.(64)”
- Cannabidiol as a Potential Treatment for Anxiety Disorders (2015, background): “[103] 15 HV DBP Oral 600 mg, acute, 1, 2, 3 h STAI scores VAMS scores ↓ STAI scores ↓ VAMS scores”