Dioxin: a review of its environmental effects and its aryl hydrocarbon receptor biology
By P. Mandal. Journal of Comparative Physiology □ B, 2005
599 citations recorded by the source corpus.
Subfields: Cancer Research, Health, Toxicology and Mutagenesis
View Paper: https://www.semanticscholar.org/paper/446a8f12df3b0ed3c7f649e52d7b53b1ad3859f6
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In Cancer Research
In Health, Toxicology and Mutagenesis
Citations in the corpus
- AN INTERACTION BETWEEN KYNURENINE AND THE ARYL HYDROCARBON RECEPTOR CAN GENERATE REGULATORY T CELLS, (2010, background): “Activation of the AHR by this environmental pollutant can lead to a range of toxic endpoints, including hepatocellular damage, epithelial changes, cancer, birth defects, thymic involution, and immunosuppression (10, 11).”
- Species Sensitivity to Toxic Substances: Evolution, Ecology and Applications (2020, background): “Gene expression changes resulting from AH-receptor mediated transcriptional activation in birds has been convincingly related to a range of pathologies linked to DNA damage, changes in hormone cycling, teratogenicity, neurotoxicity and carcinogenesis (Mandal, 2005).”
- Pollutants inducing epigenetic changes and diseases (2019, background): “Since dioxin is a weak mutagen, numerous researches have been aimed at uncovering the potential mechanisms contributing to its carcinogenicity (Mandal 2005).”
- Human health effects of air pollution. (2008, background): “, 1977), as indicated by an increase in levels of certain enzymes in the blood (see following discussion on the underlying cellular mechanisms of action), as well as gastrointestinal and liver cancer (Mandal, 2005).”