Tau phosphorylation: the therapeutic challenge for neurodegenerative disease.
By D. Hanger, B. Anderton, Wendy Noble. Trends in Molecular Medicine, 2009
950 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Physiology, Psychiatry and Mental health
View Paper: https://www.semanticscholar.org/paper/420c99059a78d87c1578ee0653351d5bcc69995e
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In Cellular and Molecular Neuroscience
In Physiology
In Psychiatry and Mental health
Citations in the corpus
- Post-translational modifications of tau protein: implications for Alzheimer's disease. (2011, positive): “threonines (41%) and only 5 are tyrosines (6%) (Buee et al., 2000; Hanger et al., 2009; Sergeant et al., 2008).”
- Physiological release of endogenous tau is stimulated by neuronal activity (2013, neutral): “These authors suggest that such increased tau phosphorylation and cleavage, as occurs in Alzheimer’s disease [18], could lead to tau accumulation in cerebrospinal fluid.”