[Age-related macular degeneration].

By Alain Ramé. La Revue de l Infirmière, 2006

5,595 citations recorded by the source corpus.

Subfields: Biochemistry, Epidemiology, Ophthalmology

View Paper: https://www.semanticscholar.org/paper/40a704c870cfd5cbd0afe272119ebb06c154cc40

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Biochemistry

1. Baowei Fei
2. A. Adamis
3. Guolan Lu
4. N. Ferrara
5. I. Bhutto
6. G. Lutty

In Epidemiology

1. Baowei Fei
2. A. Adamis
3. Guolan Lu
4. N. Ferrara
5. I. Bhutto
6. G. Lutty

In Ophthalmology

1. N. Ferrara
2. Baowei Fei
3. A. Adamis
4. Guolan Lu
5. I. Bhutto
6. G. Lutty

Citations in the corpus

• Trait-Associated SNPs Are More Likely to Be eQTLs: Annotation to Enhance Discovery from GWAS (2010, background): “For a few disorders, results of GWAS have highlighted biological contributing factors to disease that had not previously been recognized as central, such as the complement system in macular degeneration [1] or autophagy in Crohn’s disease [2–4].”
• Complement System Part II: Role in Immunity (2015, background): “The loss of central vision is associated with loss of photoreceptors and drusen formation in the retina (248).”
• Understanding age-related macular degeneration (AMD): Relationships between the photoreceptor/retinal pigment epithelium/Bruch’s membrane/choriocapillaris complex (2012, background): “If AMD is caused by inflammation, it is not a classical inflammatory disease per se but a growing body of evidence suggests that chronic, abnormal inflammatory response and immunologic events play an important role in progression of AMD (Jager et al., 2008).”
• Artificial intelligence in retina. (2018, neutral): “Despite the frequent visits, slow but irreversible visual decline may result from the frequent recurrences that are essentially required to retreat the patient (Schmidt-Erfurth et al., 2015) as well as out of potential delays between the diagnosis of an exudative recurrence and subsequent retreatmen”
• Retinal Fundus Multi-Disease Image Dataset (RFMiD): A Dataset for Multi-Disease Detection Research (2021, background): “The image is labeled as ARMD if it shows any of the following clinical findings: multiple drusen in macular region, geographic atrophy (see Figure 2b) involving the fovea, features of neovascular age-related macular degeneration [18].”
• The Impact of Oxidative Stress and Inflammation on RPE Degeneration in Non-neovascular AMD (2017, background): “Due to its high metabolic activity, RPE are enriched with mitochondria, and as a result, is a major source of ROS in the RPE (Jager et al., 2008).”
• Ten years of anti-vascular endothelial growth factor therapy (2016, background): “In addition to neovascularization and vascular leak, neovascular AMD and DME are also characterized by immune cell infiltrates and neural cell death 238 , against which anti-VEGFA drugs are not effective.”
• Endoplasmic reticulum stress: molecular mechanism and therapeutic targets (2023, background): “AMD is the third leading cause of irreversible blindness worldwide, usually affecting people aged >55 years.(259) AMD was estimated to affect 196 million people in 2020 and is expected to rise to 288 million by 2040, with the largest number of cases in Asia (113 million).”
• Risk factors for the incidence of Advanced Age-Related Macular Degeneration in the Age-Related Eye Disease Study (AREDS) AREDS report no. 19. (2003, neutral): “AMD category 2 1053 (42) 3a 1453 (58) 4a Neovascular AMD 714 Central GA 74 AREDS treatment Antioxidants 633 (25) 211 (27) Zinc 633 (25) 197 (25) Antioxidants zinc 609 (24) 192 (24) Placebo 631 (25) 188 (24) Demographic and behavioral Age (yrs) 65 548 (22) 122 (15) 65–69 888 (35) 198 (25) 70 1070 (43”