Molecular regulation of HDL metabolism and function: implications for novel therapies.

By D. Rader. Journal of Clinical Investigation, 2006

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Pathophysiology of diabetic dyslipidaemia: where are we? (2015, background): “During the catabolic process, lipid-poor ApoA-I is formed that can be filtered at the level of the glomerulus and then catabolised by proximal renal tubular epithelial cells after binding to cubilin, a protein localised to the apical surface of the renal tubular cell [13].”
Macrophage phenotypes in atherosclerosis (2014, background): “Moreover, M-CSF-derived macrophage have been referred to as CD68+ CD14+, whereas GM-CSF derived macrophages were identified as CD68+ CD14 characterized by a high expression level of genes involved in reverse cholesterol transport (PPARc, LXRa, and ABCG1) and in macrophage emigration (CCR7) (61).”