The immunology of acute stroke

By Á. Chamorro, A. Meisel, A. Planas, X. Urra, D. Beek, R. Veltkamp. Nature Reviews Neurology, 2012

674 citations recorded by the source corpus.

Subfields: Immunology, Neurology

View Paper: https://www.semanticscholar.org/paper/3d9270c3387c3b5c8e20d4418b2d94907580d070

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In Neurology

1. Martin Dichgans

Citations in the corpus

• Mechanisms of neuronal cell death in ischemic stroke and their therapeutic implications (2021, background): “inflammatory response, causing a massive influx of macrophages, neutrophil polymorphs, and lymphocytes into the brain parenchyma.(63) In the early stages of ischemic stroke, neutrophil accumulation occurs in the ischemic core, while microglial activation and proliferation occurs in the penumbra.”
• Microbiota Dysbiosis Controls the Neuroinflammatory Response after Stroke (2016, background): “…a key role for differential Thelper cell priming in stroke pathophysiology, in which proinflammatory Th1, Th17, and T cells deteriorate poststroke inflammation, whereas Treg cells limit the inflammatory collateral damage (Shichita et al., 2009; Iadecola and Anrather, 2011; Chamorro et al., 2012).”
• Ischemia/Reperfusion (2016, neutral): “transplantation (41,60,125,347,348,469,512,573,610,717), but some reports are contradictory (101-103, 458, 873).”