The immune-mediated alteration of serotonin and glutamate: towards an integrated view of depression

By N. Müller, M. Schwarz. Molecular Psychiatry, 2007

666 citations recorded by the source corpus.

Subfields: Behavioral Neuroscience, Biological Psychiatry, Social Psychology

View Paper: https://www.semanticscholar.org/paper/3c87161b5a3719563b21d3fb14179c7af164a71f

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Behavioral Neuroscience

1. Andrew H. Miller
2. Charles L Raison
3. L. Capuron
4. V. Maletić
5. Jonathan P. Godbout
6. Michael R. Irwin
7. Gregory E. Miller
8. C. Nemeroff

In Biological Psychiatry

1. Andrew H. Miller
2. Charles L Raison
3. V. Maletić
4. L. Capuron
5. Jonathan P. Godbout
6. Michael R. Irwin
7. George M. Slavich
8. C. Nemeroff

In Social Psychology

1. Andrew H. Miller
2. Charles L Raison
3. V. Maletić
4. L. Capuron
5. Michael R. Irwin
6. S. Mazmanian
7. T. Sampson
8. M. Del Giudice

Citations in the corpus

• Minocycline attenuates lipopolysaccharide (LPS)-induced neuroinflammation, sickness behavior, and anhedonia (2008, background): “IDO mediated TRP metabolism in the brain may affect behavior by impacting both serotonin and glutamate pathways [39].”
• Inflammation and Its Discontents: The Role of Cytokines in the Pathophysiology of Major Depression (2009, positive): “Quinolinic acid also induces oxidative stress, which in combination with glutamate release may contribute to CNS excitotoxicity (see below) (36,40-42).”
• Immune System to Brain Signaling: Neuropsychopharmacological Implications (2011, background): “QUIN also induces oxidative stress, which in combination with glutamate release may contribute to CNS excitotoxicity (Rios and Santamaria, 1991; Schwarcz and Pellicciari, 2002; Muller and Schwarz, 2007; McNally et al., 2008).”