Induced Disease Resistance in Plants by Chemicals

By M. Oostendorp, W. Kunz, Bob Dietrich, T. Staub. European journal of plant pathology, 2004

495 citations recorded by the source corpus.

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Citations in the corpus

Resistance to Leptosphaeria maculans (phoma stem canker) in Brassica napus (oilseed rape) induced by L. biglobosa and chemical defence activators in field and controlled environments (2006, background): “In general, the resistance induced in dicotyledonous plants by ASM is thought to last for shorter times than that induced in monocotyledonous hosts (Oostendorp et al., 2001), and further work is needed to understand the basis of the long-term resistance induced in oilseed rape.”
Biocontrol mechanisms of Trichoderma harzianum against soybean charcoal rot caused by Macrophomina phaseolina (2016, unknown): “Similar findings about the increase in POX activity after the application of biocontrol agents have been reported in different crops (Oostendorp et al. 2001).”
Water deficit effect on the accumulation of biomass and artemisinin in annual wormwood(Artemisia annua L., Asteraceae) (2010, background): “Plants have evolved a wide range of secondary metabolites that deter herbivores, pathogens, competing species, attract pollinators, and protect them from abiotic stress such as water deficit (Harborne, 1993; Baldwin et al., 2001; Oostendorp et al., 2001; Heath and Boller, 2002; Wittstock and Ghershe”
Integrated control of cotton root rot disease by mixing fungal biocontrol agents and resistance inducers (2009, background): “Certain chemicals, such as salicylic acid (SA), 2,6-dichloroisonicotinic acid (INA), potassium salts, amino butyric acid (BABA) and benzo(1,2,3) thiadiazole-7-carbothioic acid S-methyl ester (Bion) were reported to induce SAR in plants against some plant pathogens (Oostendorp et al., 2001).”
Trichoderma species — opportunistic, avirulent plant symbionts (2004, background): “Induction of resistance in plants Localized and systemic induced resistance occurs in all or most plants in response to attack by pathogenic microorganisms, physical damage due to insects or other factors, treatment with various chemical inducers and the presence of non-pathogenic RHIZOBACTERIA”