The Pancreas Cancer Microenvironment

By C. Feig, A. Gopinathan, A. Neesse, Derek S. Chan, N. Cook, D. Tuveson. Clinical Cancer Research, 2012

1,225 citations recorded by the source corpus.

Subfields: Molecular Biology, Oncology

View Paper: https://www.semanticscholar.org/paper/3919fa7faf04fc82d28d21a40bd6d37cf802c679

Citations in the corpus

• Depletion of Carcinoma-Associated Fibroblasts and Fibrosis Induces Immunosuppression and Accelerates Pancreas Cancer with Diminished Survival (2014, neutral): “The cellular component of the desmoplastic stroma in PDAC is composed primarily of myofibroblasts, characterized by aSMA expression (Feig et al., 2012; Rasheed et al., 2012).”
• Vitamin D Receptor-Mediated Stromal Reprogramming Suppresses Pancreatitis and Enhances Pancreatic Cancer Therapy (2014, neutral): “Reciprocally, activated PSCs produce PDGF, insulin-like growth factor 1 (IGF1), connective tissue growth factor (CTGF), and other factors that may promote cancer cell proliferation, survival, and migration (Apte and Wilson, 2012; Feig et al., 2012).”