Intestinal inflammation allows Salmonella to use ethanolamine to compete with the microbiota

By Parameth Thiennimitr, Sebastian E. Winter, M. Winter, M. Xavier, V. Tolstikov, D. Huseby, Torsten Sterzenbach, R. Tsolis, J. Roth, A. Bäumler. Proceedings of the National Academy of Sciences of the United States of America, 2011

618 citations recorded by the source corpus.

Subfields: Food Science, Molecular Biology, Oncology

View Paper: https://www.semanticscholar.org/paper/372a7bddfd761bdb94f456b1841b95ba5e7c2903

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Food Science

In Molecular Biology

Citations in the corpus

Dysfunction of the intestinal microbiome in inflammatory bowel disease and treatment (2012, background): “Recent studies have shown that redox stress allows Salmonella to use ethanolamine as a carbon source [80] and allows enterohemorrhagic E.”
Colonocyte metabolism shapes the gut microbiota (2018, background): “enterica a growth advantage over obligate anaerobic bacteria because it enables the pathogen to consume microbiota-derived fermentation products, such as succinate (16), butyrate (15), ethanolamine (78), or 1,2-propanediol (79).”
Depletion of butyrate-producing Clostridia from the gut microbiota drives an aerobic luminal expansion of Salmonella (2016, background): “Typhimurium during colitis is the generation of respiratory electron acceptors as a by-product of the inflammatory host response (Ali et al., 2014; Lopez et al., 2015, 2012; Rivera-Chávez et al., 2013; Thiennimitr et al., 2011; Winter et al., 2010).”
From Animalcules to an Ecosystem: Application of Ecological Concepts to the Human Microbiome (2012, background): “By stimulating an inflammation response, S. enterica is able to indirectly create a novel niche and thereby avoid competition for more commonly used redox combinations (Thiennimitr et al. 2011).”