PPAR γ insufficiency enhances osteogenesis through osteoblast formation from bone marrow progenitors

By T. Akune, Shinsuke Ohba, Satoru Kamekura, M. Yamaguchi, U. Chung, N. Kubota, Y. Terauchi, Y. Harada, Y. Azuma, Kozo Nakamura and 2 more. 2004

882 citations recorded by the source corpus.

Subfields: Molecular Biology, Orthopedics and Sports Medicine

View Paper: https://www.semanticscholar.org/paper/36d1fd751af9f67da7b7a519c7b8f7e32e139c66

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

1. R. Jilka
2. Charles A. O’Brien
3. S. Manolagas
4. S. Khosla
5. L. Bonewald

In Orthopedics and Sports Medicine

1. S. Manolagas

Citations in the corpus

• Molecular and cellular mechanisms of the anabolic effect of intermittent PTH. (2007, background): “Thus, PPARγ exerts a tonic suppressive effect on osteoblast differentiation and bone formation, most probably by diverting uncommitted progenitors into the adipocyte lineage at the expense of the osteoblast lineage.”
• Matrix-embedded cells control osteoclast formation (2011, background): “Second, a variety of genetic modifications in mice alter osteoblast number without changing osteoclast numbe”