Mild and moderate asthma is associated with airway goblet cell hyperplasia and abnormalities in mucin gene expression.

By Claudia L. Ordoñez, Ramin Khashayar, Hofer Wong, Ron Ferrando, Reen Wu, Dallas M. Hyde, Jon A. Hotchkiss, Yifan Zhang, Alexander Novikov, G. Dolganov and 1 more. American Journal of Respiratory and Critical Care Medicine, 2001

615 citations recorded by the source corpus.

Subfields: Physiology, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/3558e5753f8da30d88994ce19ecae1444c6a4d5d

How researchers see this paper

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In Physiology

1. J. Voynow
2. M. Rose

In Pulmonary and Respiratory Medicine

1. J. Voynow
2. M. Rose
3. A. Varki

Citations in the corpus

• Anti-IL-5 treatment reduces deposition of ECM proteins in the bronchial subepithelial basement membrane of mild atopic asthmatics. (2003, background): “Other features of remodeling include increased smooth muscle mass (9), goblet cell hyperplasia (10), and new blood vessel formation (11).”
• Respiratory tract mucin genes and mucin glycoproteins in health and disease. (2006, background): “MUC5AC mucin, initially isolated as tracheobronchial mucin from the lung mucus of a patient with bronchial asthma (179, 223), was subsequently shown to be present in airway secretions from both healthy individuals and asthmatic patients (116, 194, 271).”