Electrical, contractile and structural remodeling during atrial fibrillation.

By M. Allessie, J. Ausma, Ulrich Schotten. Cardiovascular Research, 2002

1,499 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine

View Paper: https://www.semanticscholar.org/paper/3072b23c2d7d1c511d7b9220a34711885b5281d4

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In Cardiology and Cardiovascular Medicine

1. Stanley Nattel
2. M. Ezekowitz
3. Gerhard Hindricks

Citations in the corpus

• 2017 HRS/EHRA/ECAS/APHRS/SOLAECE expert consensus statement on catheter and surgical ablation of atrial fibrillation (2017, background): “The transition likely reflects progressive structural and electrophysiological remodeling in both atria, making the sources of the arrhythmia more stable by fundamental mechanisms that have been incompletely explored.(153,154,155,156)”
• Atrial fibrosis: mechanisms and clinical relevance in atrial fibrillation. (2008, background): “Fibrosis is a hallmark of arrhythmogenic structural emodeling (4,5).”
• ACC/AHA/ESC 2006 guidelines for the management of patients with atrial fibrillation--executive summary: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines and the European Society of Cardiology Committee for Practice Guidelines (Writing Commit (2006, neutral): “Atrial fibrosis may precede the onset of AF,(26) and juxtaposition of patchy fibrosis with normal atrial fibers may account for nonhomogeneity of conduction.(27) Interstitial fibrosis may result from apoptosis leading to replacement of atrial myocytes,(28) loss of myofibrils, accumulation of glycoge”
• Arrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation. (2007, background): “AF alters atrial electrophysiological properties in a way that favors AF occurrence (this auto-perpetuation phenomenon has been called “AF begets AF”), both by increasing AF sustainability and by enhancing atrial vulnerability to AF induction by premature atrial beats (10, 71, 218, 342).”
• EHRA/HRS/APHRS/SOLAECE expert consensus on atrial cardiomyopathies: definition, characterization, and clinical implication. (2016, neutral): “Atrial fibrillation itself leads to remodelling, causing electrophysiological (electrical), contractile, and structural changes.(495,496) Although AF can typically be reversed A.”
• ACC/AHA/ESC 2006 guidelines for the management of patients with atrial fibrillation-executive summary: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines and the European Society of Cardiology Committee for Practice Guidelines (Writing Committ (2006, neutral): “Atrial fibrosis may precede the onset of AF (26), and juxtaposition of patchy fibrosis with normal atrial fibers may account for nonhomogeneity of conduction (27).”
• 2012 HRS/EHRA/ECAS expert consensus statement on catheter and surgical ablation of atrial fibrillation: recommendations for patient selection, procedural techniques, patient management and follow-up, definitions, endpoints, and research trial design (2012, background): “Evidence suggests that, when high-frequency atrial activation is maintained for at least 24 h, ion channel remodeling changes the electrophysiologic substrate [8, 19, 21], promoting sustained reentry and increasing the activity of triggers, further contributing to AF permanence.”