Base excision repair of oxidative DNA damage and association with cancer and aging.
By Scott Maynard, S. Schurman, Charlotte Harboe, N. C. de Souza-Pinto, V. Bohr. Carcinogenesis, 2008
659 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/3063bcc61150f895eade542c55c8a1bb10dca9b9
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Citations in the corpus
- Drug-Induced Oxidative Stress and Toxicity (2012, background): “BER recognizes and repairs oxidized bases, AP sites, DNA single-strand breaks, alkylated bases, deaminated bases, and base mismatches [4].”
- Oxidative Stress, Mitochondrial Dysfunction, and Aging (2011, background): “A transgenic line carrying a human SOD2 transgene under the control of a human β-actin promoter shows protection against hyperoxic lung injury [220], reduction in mitochondrial superoxide in hippocampal neurons, and extended lifespan as the result of increased activity of MnSOD [221].”