Phospholipase C-γ2 and Vav cooperate within signaling microclusters to propagate B cell spreading in response to membrane-bound antigen
By Michèle Weber, B. Treanor, D. Depoil, H. Shinohara, Naomi E. Harwood, M. Hikida, T. Kurosaki, F. Batista. Journal of Experimental Medicine, 2008
155 citations recorded by the source corpus.
Weber et al. 2008. J. Exp. Med. doi:10.1084/jem.20072619[OpenUrl][1][Abstract/FREE Full Text][2] [1]: {openurl}?query=rft.jtitle%253DJ.%2BExp.%2BMed.%26rft_id%253Dinfo%253Adoi%252F10.1084%252Fjem.20072619%26rft_id%253Dinfo%253Apmid%252F18362175%26rft.genre%253Darticle%26rft_val_fmt%253Dinfo%
Subfields: Genetics, Immunology
View Paper: https://www.semanticscholar.org/paper/2e096dca47978bc3ac4fee36c996484cfcf925af
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In Immunology
Citations in the corpus
- Programmed cell death 1 forms negative costimulatory microclusters that directly inhibit T cell receptor signaling by recruiting phosphatase SHP2 (2012, background): “Meanwhile, such signaling molecules were imaged at BCR microclusters in the planar bilayer system (Fleire et al., 2006; Weber et al., 2008).”