The role of apoptosis in response to photodynamic therapy: what, where, why, and how
By N. Oleinick, R. Morris, I. Belichenko. Photochemical and Photobiological Sciences, 2002
1,181 citations recorded by the source corpus.
Subfields: Biomedical Engineering, Molecular Biology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/2dd4cc26749d7444dc2ae3e0523b6ed9cae1cee8
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In Biomedical Engineering
- Michael R Hamblin
- Resham Bhattacharya
- P. Mukherjee
- S. Bhattacharyya
- Rachel A. Kudgus
- David B. Graves
In Molecular Biology
In Pulmonary and Respiratory Medicine
Citations in the corpus
- Photodynamic therapy of cancer. Basic principles and applications (2008, background): “MTs have been described to be altered after photodynamic treatments with porphyrins (Photofrin, BPD-MA), phthalocyanines (ZnPc, AlPc) and porphycenes (TPPo, PdTPPo) [29, 30, 33, 34].”
- Photodynamic therapy in the treatment of microbial infections: Basic principles and perspective applications (2006, background): “end-point of several decades of intensive investigations, which provided a wealth of information as regards the correlation between the chemical structure of photosensitizing agents (especially those belonging to the porphyrin family) and their pharmacokinetic properties, as well as on their mode of”
- Cell Death Pathways in Photodynamic Therapy of Cancer (2011, background): “PDT is thought to induce photodamage of Bcl-2 and related antiapoptotic proteins and activate the proapoptotic members of the family [35].”
- Oncologic Photodynamic Therapy: Basic Principles, Current Clinical Status and Future Directions (2017, background): “However, numerous other apoptotic signal transduction pathways that are being activated upon photodamage have been described (reviewed extensively by [24,27,28]).”
- Inorganic Nanoparticles in Cancer Therapy (2010, positive): “can oxidatively modify the neighboring lipids, proteins, amino acids and nucleic acid content of the cellular component and thereby lead to cellular damage by necrosis, and/or apoptosis (166).”
- Mechanisms in photodynamic therapy: part two-cellular signaling, cell metabolism and modes of cell death. (2005, background): “[3] Oleinick NL, Morris RL, Belichenko I.”