Life and death: metabolic rate, membrane composition, and life span of animals.

By A. Hulbert, R. Pamplona, R. Buffenstein, W. Buttemer. Physiological Reviews, 2007

891 citations recorded by the source corpus.

Subfields: Aging, Ecology, Endocrine and Autonomic Systems

View Paper: https://www.semanticscholar.org/paper/2b6536c7532bae4b61a8b037771db4ede1f2f5f6

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Citations in the corpus

Does the field of animal personality provide any new insights for behavioral ecology (2017, background): “(2010) argue that the correlation between metabolism and “personality” (and hormones and “personality”) is an example of a “pace-of-life” syndrome, which, in turn, really refers to a trade-off between longevity and metabolic rate (Hulbert et al. 2007).”
Reactive oxygen species as universal constraints in life-history evolution (2009, background): “Recently, Hulbert et al. (2007) have suggested that some exceptions to the rule might be explicable if variation documented in the fatty acid composition of cell membranes across species is taken into account.”
Oxidative Stress and Hormesis in Evolutionary Ecology and Physiology (2014, background): “Hence, it has been proposed that the macromolecular composition of a cell might be another antioxidant mechanism natural selection has shaped to make cells more resistant to reactive species (Hulbert et al. 2007; Pamplona and Barja 2007; Pamplona and Costantini 2011).”
Oxidative Stress and the Central Nervous System (2017, neutral): “Consequently, the brain, with its rich lipid content, high energy demand, and weak antioxidant capacity becomes an easy target of excessive oxidative insult (Hulbert et al., 2007).”
Nest predation research: recent findings and future perspectives (2015, background): “…(Martin et al. 2015), potentially because changes in growth rate over evolutionary time can allow evolutionary shifts in other traits, such as the fatty acid composition of mitochondrial cell membranes, which can offset physiological costs of faster metabolism and growth (e.g. Hulbert et al. 2007).”
Oxidative stress as a mediator of life history trade-offs: mechanisms, measurements and interpretation. (2009, background): “Therefore, it is more common to measure not the ROS themselves, but the damage that they cause; there is some justification for this as ultimately it is the damage caused by ROS that matters rather than the total amount of ROS produced.”
Telomeres: Linking stress and survival, ecology and evolution (2010, background): “Increasing the proton leak through the inner mitochondrial membrane via uncoupling proteins results in a net reduction in free radical production (Hulbert et al., 2007).”
From bivalves to birds : oxidative stress and longevity (2010, background): “Its low PI is a result of it having proportionately large amounts of peroxidationresistant MUFA, but low amounts of peroxidation-sensitive PUFA, particularly DHA (Hulbert et al. 2007).”
From molecules to living systems: historical and contemporary issues in oxidative stress and antioxidant ecology (2010, background): “In 2003, Hulbert further developed the hypothesis and proposed the membrane pacemaker theory of ageing, suggesting that it is variation inmembrane fatty acid composition, and the consequent differential vulnerability to lipid peroxidation, which determines lifespan: cells richer in PUFA should be mo”
Modern Biological Theories of Aging (2010, background): “[6] Hulbert AJ, Pamplona R, Buffenstein R and”