Cytokines and the brain: implications for clinical psychiatry.
By Z. Kronfol, D. Remick. American Journal of Psychiatry, 2000
803 citations recorded by the source corpus.
Subfields: Behavioral Neuroscience, Biological Psychiatry, Pharmacology
View Paper: https://www.semanticscholar.org/paper/2b24b32ba294a53c6073fec7840d1cbe3c621ad2
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Behavioral Neuroscience
In Biological Psychiatry
- J. Wingfield
- J. Koolhaas
- S. Korte
- Michael Berk
- S. Dickerson
- J. Kiecolt-Glaser
- R. Glaser
- Michael Maes
In Pharmacology
Citations in the corpus
- So depression is an inflammatory disease, but where does the inflammation come from? (2013, positive): “As such, periodontal disease may be a marker of a failure of the immune system to resolve inflammation [187,188], a state that may also result in vulnerability to depression [189].”
- Evidence for a cytokine model of cognitive function. (2009, background): “, 2004) and in neuropsychiatric disorders, such as depression (Anisman et al., 2008; Capuron and Dantzer, 2003; Dantzer et al., 2008; Irwin and Miller, 2007; Kronfol and Remick, 2000; Raison et al., 2006).”
- When the social self is threatened: shame, physiology, and health. (2004, background): “Chronic or repeated activation of the HPA and proinflammatory cytokine network may increase susceptibility to disease or moderate disease course (e.g., McEwen, 1998; Kronfol & Remick, 2000).”
- Major depression after breast cancer: a review of epidemiology and treatment. (2008, background): “Proinflammatory cytokines released in response to the tissue damage that occurs during cancer treatment may contribute to the high rates of depression and related symptoms in cancer patients [176,177,185–188].”
- The Darwinian concept of stress: benefits of allostasis and costs of allostatic load and the trade-offs in health and disease. (2005, background): “In animals it is generally accepted that proinflammatory cytokines, such as IL-1beta, IFN-alpha, IFN-gamma and tumor necrosis factor-alpha (TNF-alpha) induce ‘sickness behavior’ which might resemble the vegetative symptoms of depression in humans [216,217].”
- Emotions, morbidity, and mortality: new perspectives from psychoneuroimmunology. (2002, background): “The relevance of cytokines to the biobehavioral sciences is illustrated by the appearance of reviews of cytokine biology in psychiatric and psychological literature (Kronfol & Remick 2000, Maier & Watkins 1998).”