Glucocorticoids, prenatal stress and the programming of disease.
By Anjanette P. Harris, J. Seckl. Hormones and Behavior, 2011
857 citations recorded by the source corpus.
Subfields: Behavioral Neuroscience, Pediatrics, Perinatology and Child Health, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/2ace02301b4561ffb91f1f1742e38b268d5b0f93
How researchers see this paper
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In Behavioral Neuroscience
- Jens C. Pruessner
- Curt A. Sandman
- Elysia Poggi Davis
- J. Cidlowski
- T. Bale
- B. V. D. Van den Bergh
- G. Koob
In Pediatrics, Perinatology and Child Health
- Alexander Meissner
- Curt A. Sandman
- Elysia Poggi Davis
- Tessa J. Roseboom
- T. Bale
- B. V. D. Van den Bergh
- M. Weinstock
Citations in the corpus
- Paternal stress exposure alters sperm microRNA content and reprograms offspring HPA stress axis regulation (2013, background): “Interestingly, rodents models have previously demonstrated offspring HPA axis dysregulation following maternal or paternal exposures to diverse challenges including alcohol, lead, chronic stress, or social defeat, suggesting that the HPA axis is especially sensitive to environmental manipulations—th”
- Paternally-induced transgenerational environmental reprogramming of metabolic gene expression in mammals (2010, background): “…(Horton, 2005), cultural inheritance of stress reactivity and maternal grooming behavior in rats (Meaney et al., 2007; Weaver et al., 2004), and metabolic and psychiatric sequelae of fetal malnutrition in humans and rodents (Hales and Barker, 2001; Harris and Seckl, 2010; Symonds et al., 2009).”
- Prenatal developmental origins of behavior and mental health: The influence of maternal stress in pregnancy. (2017, background): “80-90 % of maternal cortisol to the inactive form: cortisone (Harris and Seckl, 2011).”
- Congenital adrenal hyperplasia due to steroid 21-hydroxylase deficiency: an Endocrine Society clinical practice guideline. (2010, neutral): “malnutrition with exposure to GCs (119).”
- Glucocorticoid receptor signaling in health and disease (2013, background): “GR−/− neonates die soon after birth due to respiratory failure arising from impaired lung development, indicating the important role of GR signaling in lung maturation [41, 42].”
- Maternal cortisol over the course of pregnancy and subsequent child amygdala and hippocampus volumes and affective problems (2012, background): “First, cortisol, the end-product of the hypothalamic–pituitary–adrenal (HPA) axis, is known to exert a wide array of metabolic, endocrine, and immune effects on most if not all cells (19, 20), and is known to play a key role in events underlying the development of the brain and other organ systems (”