BDNF-based synaptic repair as a disease-modifying strategy for neurodegenerative diseases

By B. Lu, G. Nagappan, X. Guan, Pradeep J. Nathan, P. Wren. Nature Reviews Neuroscience, 2013

736 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Physiology

View Paper: https://www.semanticscholar.org/paper/289176fb9beb8de0b4f778751e3171b612fba2c9

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. M. Chao

In Physiology

1. Bin Zhao
2. Zhou Liu
3. Chunmei Jiang
4. Pengru Huang
5. Guangning Li

Citations in the corpus

• Exercise promotes the expression of brain derived neurotrophic factor (BDNF) through the action of the ketone body β-hydroxybutyrate (2016, background): “…behavior through increased levels of hippocampal BDNF, which can enhance plasticity and synaptogenesis and reduce neurodegeneration (Aguiar et al., 2011; Cotman et al., 2007; Duman and Monteggia, 2006; Lu et al., 2013; Marais et al., 2009; Martinowich et al., 2007; Russo-Neustadt et al., 2000).”
• The Gut Microbiota and Alzheimer’s Disease (2017, positive): “BDNFisa neurotrophinthatiscrucialforsynapticplasticity andcognitivefunctionandhasbeenfoundtobe decreasedinthebrainsandseraofADpatientsand tocorrelatewithA (cid:2) burden [42–44] (for review, see [42, 44]).”