Persistence of the Epstein-Barr virus and the origins of associated lymphomas.
By D. Thorley-Lawson, A. Gross. New England Journal of Medicine, 2004
1,083 citations recorded by the source corpus.
Subfields: Infectious Diseases, Oncology
View Paper: https://www.semanticscholar.org/paper/279a505ea72252d698e0fa93b7091d98a4e628eb
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In Infectious Diseases
In Oncology
Citations in the corpus
- Mechanisms of B-cell lymphoma pathogenesis (2005, background): “MALT lymphoma API2–MALT1 (30)135, CD95 (5-80)25,140,141‡ Indirect role of – BCL10–IgH (5)136,137, Helicobactor pylori in MALT1–IgH (15-20)138, gastric MALT FOXP1–IgH (10)139 lymphomas85”
- Meningeal inflammation is widespread and linked to cortical pathology in multiple sclerosis. (2011, background): “Epstein–Barr virus infects naı¨ve B cells, which through the expression of viral proteins can undergo germinal centre-like reactions to generate long-lived memory B cells without the need for antigen or T cell co-stimulation (Thorley-Lawson and Gross, 2004 ).”