Regulation of interferon-gamma during innate and adaptive immune responses.

By J. Schoenborn, Christopher B Wilson. Advances in Immunology, 2007

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The microRNA miR-29 controls innate and adaptive immune responses to intracellular bacterial infection by targeting interferon-γ (2011, background): “The canonical T helper type 1 (T H 1) cytokine interferon-γ (IFN-γ) is critical for innate and adaptive immunity to infection, especially intracellular bacterial infections such as with Listeria monocytogenes or Mycobacterium tuberculosis”
An immune-active tumor microenvironment favors clinical response to ipilimumab (2012, background): “Many of these immune markers (such as CXCL9, CXCL10, and CXCL11) were IFN- -inducible genes [39, 40].”
Protein-binding assays in biological liquids using microscale thermophoresis. (2010, background): “hIFN-γ is a dimerized soluble cytokine that is crucial for immunity against intracellular pathogens and also for tumour contro”
CXCL9, CXCL10, CXCL11/CXCR3 axis for immune activation - a target for novel cancer therapy (2017, background): “[22] CXCL11, also known as interferon-inducible T-cell alpha chemoattractant (I-TAC) or interferon-gamma-inducible protein 9 (IP-9), is induced by IFN-γ and IFN-β, and weakly by IFN-α.”
Multiple Sclerosis: Pathogenesis, Symptoms, Diagnoses and Cell-Based Therapy (2016, background): “These cytokines are produced by Th1 cells (17).”