Transcriptional Regulation of the Human Tumor Suppressor DOK1 by E2F1
By M. Siouda, Jiping Yue, R. Shukla, S. Guillermier, Zdenko Herceg, M. Creveaux, R. Accardi, M. Tommasino, B. Sylla. 2012
22 citations recorded by the source corpus.
Subfields: Molecular Biology, Oncology
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In Molecular Biology
Citations in the corpus
- EgoNet: identification of human disease ego-network modules (2014, background): “For example, overexpression of NCOA3, especially in conjunction with high levels EGF receptor (EGFR) and HER2 (ERBB2), is associated with poor outcome after tamoxifen treatment [53,54].”
- BRK Targets Dok1 for Ubiquitin-Mediated Proteasomal Degradation to Promote Cell Proliferation and Migration (2014, background): “In addition, higher expression levels of Dok1 were detected in serous epithelial ovarian cancer as compared to normal tissues and this overexpression significantly correlated with disease-free survival of serous epithelial ovarian cancer patients [85].”