ADAM‐15: a metalloprotease that mediates inflammation
By Laetitia Charrier‐Hisamuddin, C. Laboisse, D. Merlin. 2008
80 citations recorded by the source corpus.
Subfields: Immunology and Allergy, Oncology
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Citations in the corpus
- ADAM15 regulates endothelial permeability and neutrophil migration via Src/ERK1/2 signalling. (2010, background): “Moreover, ADAM15 is upregulated in cells treated with pro-inflammatory cytokines(23) and in tissues of inflammatory diseases such as atherosclerosis,(24) rheumatoid arthritis,(25) and irritable bowels,(26) indicating the possibility that increased ADAM15 serves as an inflammatory mediator.(11) Howev”
- ADAM15 contributes to atherosclerosis by mediating endothelial barrier dysfunction via Src family kinase activity (2012, background): “Since the initial report of ADAM15 expression in human endothelial cells (ECs)(10) and animal micro/macrovessels,(11) evidence has emerged for a role of this molecule in regulating cell–cell adhesion(12) and vascular functions.(8,9,11) In particular, ADAM15 supports lung cancer metastasis by promoti”