Nitric oxide as modulator of neuronal function.
By H. Prast, A. Philippu. Progress in neurobiology, 2001
963 citations recorded by the source corpus.
Subfields: Molecular Biology, Physiology, Social Psychology
View Paper: https://www.semanticscholar.org/paper/1d5911a6805410182320d7a5344a01b03966f438
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Citations in the corpus
- Histamine in the nervous system. (2008, background): “Other effector pathways of H1R include production of arachidonic acid (AA), nitric oxide (NO), and cGMP (395, 588, 611, 691) through pertussis toxinsensitive Gi/Go protein-mediated activation of phospholipase A2 (PLA2), [Ca 2 ]i-dependent NO synthases, and NO-dependent guanylate cyclases (GC), respe”
- The physiology and pathophysiology of nitric oxide in the brain. (2005, background): “NO is a neurotransmitter and/or neuromodulator in both central and peripheral nervous systems by cGMP-dependent mechanisms (Bredt and Snyder, 1994a; Prast and Philippu, 2001; Lewko and Stepinski, 2002; Trabace and Kendrick, 2000).”
- Regulation of NMDA Receptor Subunit Expression and Its Implications for LTD, LTP, and Metaplasticity (2008, background): “[PubMed: 17196527] Steele PM, Mauk MD. Inhibitory control of LTP and LTD: stability of synapse strength.”
- The Endothelium and Its Role in Regulating Vascular Tone (2010, background): “Three isoforms of NOS exist: neuronal isoform (nNOS) which produces NO to act as a neuronal messenger that regulates synaptic neurotransmitter release [17], macrophage or inducible isoform (iNOS) which is only expressed in cells that have been exposed to inflammatory mediators or other injurious sti”
- Concepts of neural nitric oxide-mediated transmission (2008, background): “Similarly, based on in vivo sampling of the extracellular fluid, myriad interactions with other transmitters in the brain are to be expected (reviewed in Prast & Philippu, 2001), although details of what may be happening at the level of the synapse are sparse.”