Central nervous system injury-induced immune deficiency syndrome
By C. Meisel, J. Schwab, K. Prass, A. Meisel, U. Dirnagl. Nature Reviews Neuroscience, 2005
910 citations recorded by the source corpus.
Subfields: Clinical Psychology, Critical Care and Intensive Care Medicine, Neurology
View Paper: https://www.semanticscholar.org/paper/1a865ba1d46e688780dd1f811547b49ae71bbcb1
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In Neurology
- C. Iadecola
- Martin Dichgans
- J. Anrather
- S. Rivest
- J. Dreier
- Berislav Zlokovic
- Hugo D. Critchley
- Neil A. Harrison
Citations in the corpus
- Regulatory T cells are key cerebroprotective immunomodulators in acute experimental stroke (2009, background): “Severe brain ischemia also perturbs innate and adaptive immune cells, resulting in systemic immunodepression that predisposes patients after stroke to life-threatening infection”
- Microbiota Dysbiosis Controls the Neuroinflammatory Response after Stroke (2016, background): “Previous reports from our group and others have identified dysregulation of the autonomous nervous system and a pronounced stress response after stroke as one of the key pathways involved in mediating systemic effects on remote organ function after acute brain injury (Meisel et al., 2005; Chamorro e”
- Inflammation and Stroke: An Overview (2016, background): “However, this early activation of the immune system is superseded by a state of systemic immunodepression that predisposes to poststroke infections [27, 28].”
- The role of spreading depression, spreading depolarization and spreading ischemia in neurological disease (2011, background): “Thus, they could interfere with consciousness, respiratory drive, plasticity, repair, regeneration or with the other two supersystems of the body, the cardiovascular and the immune system, for example, by promoting cardiac arrhythmia or immunodepression which complicate the course of cerebral injur”
- The immunology of stroke: from mechanisms to translation (2011, background): “In turn, the ischemic brain, through the autonomic nervous system, exerts a potent suppressive effect on lymphoid organs that promotes intercurrent infections, a major determinant of stroke morbidity and mortalit”