An Overlapping Protein-Coding Region in Influenza A Virus Segment 3 Modulates the Host Response
By B. W. Jagger, H. Wise, J. Kash, K. Walters, N. Wills, Yongli Xiao, R. Dunfee, L. M. Schwartzman, A. Ozinsky, G. L. Bell and 7 more. Science, 2012
642 citations recorded by the source corpus.
Subfields: Epidemiology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/19da5f03053b4754aa897609f2bae573343faaa2
How researchers see this paper
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In Epidemiology
In Molecular Biology
- Ian A. Wilson
- Edward C. Holmes
- Richard H. Scheuermann
- Christian M. Zmasek
- Yun Zhang
- David S. Wishart
- Peer Bork
- Ivica Letunic
Citations in the corpus
- Ribosomal frameshifting and transcriptional slippage: From genetic steganography and cryptography to adventitious use (2016, background): “inflammatory, apoptotic and T lymphocyte-signaling pathways (39).”
- Influenza Research Database: An integrated bioinformatics resource for influenza virus research (2016, background): “In recent years, the influenza community has identified several novel proteins generated from non-canonical translation strategies such as leaky ribosomal scanning (PB1-F2 (10), PB1-N40 (11), PA-N155 (12) and PA-N182 (12)), ribosomal frameshift (PA-X (13)) and alternative splicing (M42 (14) and NS3 ”
- New World Bats Harbor Diverse Influenza A Viruses (2013, positive): “Key structural features of the Peruvian bat influenza virus genome, including RNA transcription and replication promoter elements, open reading frames and mRNA splice signals, and ribosomal frameshift element (PA gene), are nearly identical to the recently described Guatemalan bat influenza genomes ”