The right ventricle under pressure: cellular and molecular mechanisms of right-heart failure in pulmonary hypertension.
By H. Bogaard, K. Abe, A. Vonk Noordegraaf, N. Voelkel. Chest, 2009
689 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Pulmonary and Respiratory Medicine, Surgery
View Paper: https://www.semanticscholar.org/paper/1953027aa1ee86f38b82551d0f9dc56d757a53e3
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Citations in the corpus
- Anatomy, Function, and Dysfunction of the Right Ventricle: JACC State-of-the-Art Review. (2019, background): “Cardiomyocyte hypertrophy occurs through accumulation of sarcomeric proteins, usually accompanied by the re-emergence of a fetal gene expression pattern such as increased expression of natriuretic peptides and a switch from a-myosin to b-myosin heavy chain that exhibits reduced energy requirements b”
- Mechanisms of disease: pulmonary arterial hypertension (2011, background): “177 The increase in wall stress also impedes myocardial perfusion; impaired right coronary artery systolic flow and right ventricular ischemia have both been demon strated in patients with PAH. 180, 181 Mechanical stretch is translated into intracellular signaling pathways via integrins, 182 and int”