Transcallosal inhibition in chronic subcortical stroke

344 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Dynamic causal modeling of cortical activity from the acute to the chronic stage after stroke (2011, neutral): “TMS studies on interhemispheric inhibition suggested that contralesionalM1 exerts enhanced inhibitory influences on ipsilesional M1 in some patients, thereby contributing to the motor deficit (Duque et al., 2005; Murase et al., 2004).”
Breaking the silence: brain-computer interfaces (BCI) for communication and motor control. (2006, neutral): “Duque et al .”
Modulating cortical connectivity in stroke patients by rTMS assessed with fMRI and dynamic causal modeling (2010, positive): “Furthermore, it remains unclear why a subcortical lesion in the depth of the white matter may trigger pathological transcallosal interactions among the hemispheres (Duque et al., 2005; Murase et al., 2004).”