Müller cells in the healthy and diseased retina.
By A. Bringmann, T. Pannicke, J. Grosche, M. Francke, P. Wiedemann, S. Skatchkov, Neville N. Osborne, André Reichenbach. Progress in retinal and eye research, 2010
1,776 citations recorded by the source corpus.
Subfields: Molecular Biology, Ophthalmology, Physiology
View Paper: https://www.semanticscholar.org/paper/179be4e0eb95a6a6476d203cdff1fc01358920ae
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In Molecular Biology
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In Physiology
Citations in the corpus
- New functions of Müller cells (2013, background): “Müller cells interact with the neurons of their columns in a kind of symbiotic relationship (Bringmann et al., 2006; Reichenbach and Bringmann, 2010).”
- Astrocytes: biology and pathology (2009, background): “It is not clear whether the reactive gliosis in the retina is merely a response to a process afflicting the ganglion neurons or whether the gliosis contributes to that degeneration by loss of glial supportive functions, such as glutamate uptake, or contribution of degenerative effects such as produc”
- Physiology of Astroglia. (2018, neutral): “The side processes of Müller cells contact and enwrap neurons (some of these processes have velate morphology), cover synapses (by fine perisynaptic processes), and form endfeet, which plaster blood vessels (1447, 1450).”
- Late-Stage Neuronal Progenitors in the Retina Are Radial Müller Glia That Function as Retinal Stem Cells (2007, background): “Key words: retinal stem cells; Müller glia; photoreceptors; regeneration; zebrafish; radial glia”
- The biophysics of neuronal growth (2010, background): “the maintenance of the extracellular homeostasis [9], help to”
- Molecular basis of the inner blood-retinal barrier and its breakdown in diabetic macular edema and other pathological conditions. (2013, background): “Under pathological conditions, glial cells are activated and overexpress angiogenic cytokines such as VEGF, basic fibroblast growth factor, TNF-a and MMPs (Behzadian et al., 2001; Bringmann et al., 2006).”