Apoptotic mechanisms after cerebral ischemia.
By B. Broughton, D. Reutens, C. Sobey. Stroke, 2009
1,204 citations recorded by the source corpus.
Subfields: Immunology, Molecular Biology, Neurology
View Paper: https://www.semanticscholar.org/paper/1781c5653694aae3d12fef54c776f8d14ebc439f
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Immunology
In Molecular Biology
Citations in the corpus
- Neuronal Cell Death. (2018, neutral): “The penumbra may comprise as much as half the total lesion volume (71) and may undergo dynamic changes.”
- Cell Biology of Ischemia/Reperfusion Injury (2012, background): “Caspase-3 acts as the cell’s executioner by proteolyzing many cellular proteins (Broughton et al., 2009; Kroemer et al., 2007; Whelan et al., 2010).”
- Ischemia/Reperfusion (2016, neutral): “Lastly, DNA fragmentation characteristic of this form of apoptosis is mediated by endo-G (94, 453, 843).”