Galectin-3 Marks Activated Macrophages in Failure-Prone Hypertrophied Hearts and Contributes to Cardiac Dysfunction
By U. Sharma, Saraswati Pokharel, T. van Brakel, Jop H. van Berlo, J. Cleutjens, B. Schroen, S. André, H. Crijns, H. Gabius, J. Maessen and 1 more. Circulation, 2004
924 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Immunology
View Paper: https://www.semanticscholar.org/paper/168179b9f51d965df19a3f1f2d16cd12616c5292
How researchers see this paper
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In Cardiology and Cardiovascular Medicine
- Ramachandran S. Vasan
- Daniel Levy
- Michael J. Pencina
- Martin G. Larson
- Véronique L. Roger
- Jay P. Singh
- D. Lloyd‐Jones
- E. Lakatta
Citations in the corpus
- Galectin-3, a Marker of Cardiac Fibrosis, Predicts Incident Heart Failure in the Community (2012, background): “Galectin-3 (Gal-3) is a beta-galactoside-binding lectin that appears to be a mediator of cardiac fibrosis in a number of recent experimental studies (6,7).”