Insecticide resistance in insect vectors of human disease.
By J. Hemingway, H. Ranson. Annual Review of Entomology, 2000
1,573 citations recorded by the source corpus.
Subfields: Molecular Biology, Plant Science, Public Health, Environmental and Occupational Health
View Paper: https://www.semanticscholar.org/paper/1577e3df095c1991fd2832bcf334ac542a3c470e
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Citations in the corpus
- Reducing Plasmodium falciparum Malaria Transmission in Africa: A Model-Based Evaluation of Intervention Strategies (2010, background): “DDT resistance at varying levels has now been reported in over 50 anopheline species [89]; thus, to reduce the further emergence of resistance, elimination campaigns should aim to reduce transmission as rapidly as possible.”
- Conserving the efficacy of insecticides against Plutella xylostella (L.) (Lep., Plutellidae) (2005, background): “The insensitivity of c-acetylbutyric acid (GABA) receptor, which is a hetromultimeric gated chloride-ion channel in insects, is also known to be involved in resistance to certain insecticides such as avermectins, pyrethroids and cyclodienes (Hemingway and Ransom, 2000).”
- Point mutations in domain II of the voltage-gated sodium channel gene in deltamethrin-resistant Aedes aegypti (Diptera: Culicidae) (2010, background): “Many reports have indicated that the progressive use of pyrethroid insecticides has led to the development of resistance in many pest insects, including mosquitoes (Hemingway and Ranson, 2000).”
- Contemporary status of insecticide resistance in the major Aedes vectors of arboviruses infecting humans (2017, background): “Whilst the mode of action is unclear for most of these genes, CCEs catalyse the hydrolysis of ester bonds and have been frequently linked to both organophosphate and carbamate resistance, either through insecticide sequestration or direct metabolism [58].”