Effects of insulin resistance and type 2 diabetes on lipoprotein subclass particle size and concentration determined by nuclear magnetic resonance.
By W. Garvey, Soonho Kwon, D. Zheng, S. Shaughnessy, P. Wallace, A. Hutto, Kim B Pugh, A. Jenkins, R. Klein, Y. Liao and 1 more. Diabetes, 2003
608 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/1510c68f1d9fda3ff2a0e4f2f841fa996770e36e
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In Endocrinology, Diabetes and Metabolism
In Epidemiology
In Molecular Biology
Citations in the corpus
- Definition and diagnostic criteria of clinical obesity (2025, unknown): “119 The increase in the concentration of circulating large triglyceride-containing VLDL molecules found in obesity is due to greater hepatic production driven by increased fatty acid flux to the liver (often linked to excess liver fat) and reduced clearance due to decreased lipoprotein lipase.”
- Functionally Defective High-Density Lipoprotein: A New Therapeutic Target at the Crossroads of Dyslipidemia, Inflammation, and Atherosclerosis (2006, background): “In obesity and insulin resistance, frequent features of both MetS and type 2 diabetes, plasma levels of large HDL decrease in parallel with those of HDL-C, whereas levels of small HDL do not (Garvey et al., 2003; Festa et al., 2005; Goff et al., 2005; Okazaki et al., 2005).”
- Pathophysiology of diabetic dyslipidaemia: where are we? (2015, background): “The characteristic hypertriglyceridaemia observed in patients with type 2 diabetes stimulates CETP, leading to the preferential formation of triacylglycerol-rich small, dense LDL particles over larger ones [74].”