Immunohistochemical Spectrum of GISTs at Different Sites and Their Differential Diagnosis with a Reference to CD117 (KIT)
By M. Miettinen, Leslie Sobin, M. Sarlomo‐Rikala. Modern Pathology, 2000
615 citations recorded by the source corpus.
Subfields: Gastroenterology, Neurology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/113f98022951e9c9f4d4b084429c25cae47893fd
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In Gastroenterology
Citations in the corpus
- Inflammatory myofibroblastic tumours: where are we now? (2007, background): “11 84 Other entities to consider in the differential diagnosis of cellular IMTs are dendritic cell neoplasms, which character-istically show an evenly distributed chronic inflammatory infiltrate admixed with the spindle cell component.”
- KIT activation is a ubiquitous feature of gastrointestinal stromal tumors. (2001, background): “The generalized nature of KIT oncogenic activation and the evidence for a key KIT role in GIST pathogenesis are supported by immunohistochemical correlations and developmental biology evidence as follows: (a) KIT expression, as determined by immunohistochemistry, is diffuse and strong in virtually a”
- Gastrointestinal Stromal Tumors of the Stomach: A Clinicopathologic, Immunohistochemical, and Molecular Genetic Study of 1765 Cases With Long-term Follow-up (2005, neutral): “Greater curvature (gastric body) 113 59:54 (52) 68 (28–90) 4-20-17-27 0-4-5-23 Epithelioid nonmalignant 37% 15/60 (25) 33/40 (83) 34/47 (72) 9/50 (18) 2/48 (4) 0/50 (0)”