Risk stratification in the long-QT syndrome.
By S. Priori, P. Schwartz, C. Napolitano, R. Bloise, E. Ronchetti, Massimiliano Grillo, A. Vicentini, C. Spazzolini, J. Nastoli, Georgia Bottelli and 2 more. New England Journal of Medicine, 2003
760 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/0ee74196fd8599bbc266608f75bc6bf795fc971f
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In Cardiology and Cardiovascular Medicine
- Bernard R. Chaitman
- C. Semsarian
- Rick A. Nishimura
- J. Halperin
- Jose Luis Zamorano
- J. Towbin
- Douglas P. Zipes
In Molecular Biology
Citations in the corpus
- ACC/AHA/ESC 2006 guidelines for management of patients with ventricular arrhythmias and the prevention of sudden cardiac death: A report of the American College of Cardiology/American Heart Association Task Force and the European Society of Cardiology Committee for Practice Guidelines (2006, background): “In patients affected by LQTS, genetic analysis is useful for risk stratification (199) and for making therapeutic decisions (204).”
- HRS/EHRA expert consensus statement on the state of genetic testing for the channelopathies and cardiomyopathies: this document was developed as a partnership between the Heart Rhythm Society (HRS) and the European Heart Rhythm Association (EHRA). (2011, background): “LQTS is more likely to express itself efore puberty in males and after puberty in females.(17,25,26) Besides age and sex, the degree of QTc prolongation is associated with likelihood of a first LQT-triggered cardiac event (syncope or aborted cardiac arrest) while occurrence of such cardiac events, p”