The role of inflammatory cytokines in diabetic nephropathy.
By J. Navarro-González, C. Mora-Fernández. Journal of the American Society of Nephrology, 2008
917 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology
View Paper: https://www.semanticscholar.org/paper/09e8925d8d4e354698ac21edaf1f44f6bf33b894
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Citations in the corpus
- Relationship between oxidative stress and inflammatory cytokines in diabetic nephropathy. (2012, background): “IL-1 increases the expression of chemotactic factors and adhesion molecules, enhances vascular endothelial permeability, and stimulates the proliferation of mesangial cells and matrix synthesis [53,54].”
- Mechanisms of diabetic complications. (2013, background): “Circulating concentrations of TNF- are elevated in individuals with either type 1 or type 2 diabetes compared with healthy control subjects (323, 418).”
- Inflammatory molecules and pathways in the pathogenesis of diabetic nephropathy (2011, background): “Today, growing evidence indicates that these cytokines are important in the pathogenesis of microvascular complications in patients with diabetes mellitus, including diabetic nephropathy ( Box 2 )”